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The Sense and Sensibility of Strand Exchange in Recombination Homeostasis


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Vyšlo v časopise: The Sense and Sensibility of Strand Exchange in Recombination Homeostasis. PLoS Genet 10(1): e32767. doi:10.1371/journal.pgen.1004104
Kategorie: Perspective
prolekare.web.journal.doi_sk: https://doi.org/10.1371/journal.pgen.1004104

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Zdroje

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2. LiuY, GainesWA, CallenderT, BusyginaV, OkeA, et al. (2014) Down-regulation of Rad51 activity during meiosis in yeast prevents competition with Dmc1 for repair of double-strand breaks. PLoS Genet 10: e1004005 doi:10.1371/journal.pgen.1004005

3. Hunter N (2007) Meiotic recombination. In: Aguilera A, Rothstein R, editors. Topics in current genetics, molecular genetics of recombination. Heidelberg: Springer-Verlag. pp. 381–442.

4. GlobusST, KeeneyS (2012) The joy of six: how to control your crossovers. Cell 149: 11–12.

5. JoyceEF, PedersenM, TiongS, White-BrownSK, PaulA, et al. (2011) Drosophila ATM and ATR have distinct activities in the regulation of meiotic DNA damage and repair. J Cell Biol 195: 359–367.

6. LangeJ, PanJ, ColeF, ThelenMP, JasinM, et al. (2011) ATM controls meiotic double-strand-break formation. Nature 479: 237–240.

7. ZhangL, KlecknerNE, StorlazziA, KimKP (2011) Meiotic double-strand breaks occur once per pair of (sister) chromatids and, via Mec1/ATR and Tel1/ATM, once per quartet of chromatids. Proc Natl Acad Sci U S A 108: 20036–20041.

8. MartiniE, DiazRL, HunterN, KeeneyS (2006) Crossover homeostasis in yeast meiosis. Cell 126: 285–295.

9. CloudV, ChanYL, GrubbJ, BudkeB, BishopDK (2012) Rad51 is an accessory factor for Dmc1-mediated joint molecule formation during meiosis. Science 337: 1222–1225.

10. GrayS, AllisonRM, GarciaV, GoldmanAS, NealeMJ (2013) Positive regulation of meiotic DNA double-strand break formation by activation of the DNA damage checkpoint kinase Mec1(ATR). Open Biol 3: 130019.

11. RockmillB, LefrançoisP, Voelkel-MeimanK, OkeA, RoederGS, et al. (2013) High throughput sequencing reveals alterations in the recombination signatures with diminishing spo11 activity. PLoS Genet 9: e1003932.

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Štítky
Genetika Reprodukčná medicína

Článok vyšiel v časopise

PLOS Genetics


2014 Číslo 1
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