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preLekára.sk / Odborné časopisy / PLOS Pathogens / 2011 - 9

Exploiting and Subverting Tor Signaling in the Pathogenesis of Fungi, Parasites, and Viruses

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Vyšlo v časopise: Exploiting and Subverting Tor Signaling in the Pathogenesis of Fungi, Parasites, and Viruses. PLoS Pathog 7(9): e32767. doi:10.1371/journal.ppat.1002269
Kategorie: Pearls
prolekare.web.journal.doi_sk: https://doi.org/10.1371/journal.ppat.1002269

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Zdroje

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2. ShertzCABastidasRJLiWHeitmanJCardenasME 2010 Conservation, duplication, and loss of the Tor signaling pathway in the fungal kingdom. BMC Genomics 11 510

3. Madeira da SilvaLBeverleySM 2010 Expansion of the target of rapamycin (TOR) kinase family and function in Leishmania shows that TOR3 is required for acidocalcisome biogenesis and animal infectivity. Proc Natl Acad Sci U S A 107 11965 11970

4. BarquillaACrespoJLNavarroM 2008 Rapamycin inhibits trypanosome cell growth by preventing TOR complex 2 formation. Proc Natl Acad Sci U S A 105 14579 14584

5. De VirgilioCLoewithR 2006 The TOR signalling network from yeast to man. Int J Biochem Cell Biol 38 1476 1481

6. LoewithRJacintoEWullschlegerSLorbergACrespoJL 2002 Two TOR complexes, only one of which is rapamycin sensitive, have distinct roles in cell growth control. Mol Cell 10 457 468

7. BastidasRJHeitmanJCardenasME 2009 The protein kinase Tor1 regulates adhesin gene expression in Candida albicans. PLoS Pathog 5 e1000294 doi:10.1371/journal.ppat.1000294

8. HoyerLLGreenCBOhSHZhaoX 2008 Discovering the secrets of the Candida albicans agglutinin-like sequence (ALS) gene family–a sticky pursuit. Med Mycol 46 1 15

9. DavisDABrunoVMLozaLFillerSGMitchellAP 2002 Candida albicans Mds3p, a conserved regulator of pH responses and virulence identified through insertional mutagenesis. Genetics 162 1573 1581

10. RichardMLNobileCJBrunoVMMitchellAP 2005 Candida albicans biofilm-defective mutants. Eukaryot Cell 4 1493 1502

11. ZacchiLFGomez-RajaJDavisDA 2010 Mds3 regulates morphogenesis in Candida albicans through the TOR pathway. Mol Cell Biol 30 3695 3710

12. FigueiredoRCRosaDSSoaresMJ 2000 Differentiation of Trypanosoma cruzi epimastigotes: metacyclogenesis and adhesion to substrate are triggered by nutritional stress. J Parasitol 86 1213 1218

13. FennKMatthewsKR 2007 The cell biology of Trypanosoma brucei differentiation. Curr Opin Microbiol 10 539 546

14. BarquillaANavarroM 2009 Trypanosome TOR as a major regulator of cell growth and autophagy. Autophagy 5 256 258

15. BarquillaANavarroM 2009 Trypanosome TOR complex 2 functions in cytokinesis. Cell Cycle 8 697 699

16. CaoWManicassamySTangHKasturiSPPiraniA 2008 Toll-like receptor-mediated induction of type I interferon in plasmacytoid dendritic cells requires the rapamycin-sensitive PI(3)K-mTOR-p70S6K pathway. Nat Immunol 9 1157 1164

17. BogdanCMattnerJSchleicherU 2004 The role of type I interferons in non-viral infections. Immunol Rev 202 33 48

18. JaramilloMGomezMALarssonOShioMTTopisirovicI 2011 Leishmania repression of host translation through mTOR cleavage is required for parasite survival and infection. Cell Host Microbe 9 331 341

19. GingrasACRaughtBSonenbergN 2004 mTOR signaling to translation. Curr Top Microbiol Immunol 279 169 197

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21. FingarDCBlenisJ 2004 Target of rapamycin (TOR): an integrator of nutrient and growth factor signals and coordinator of cell growth and cell cycle progression. Oncogene 23 3151 3171

22. MoodyCAScottRSAmirghahariNNathanCOYoungLS 2005 Modulation of the cell growth regulator mTOR by Epstein-Barr virus-encoded LMP2A. J Virol 79 5499 5506

23. SodhiAChaisuparatRHuJRamsdellAKManningBD 2006 The TSC2/mTOR pathway drives endothelial cell transformation induced by the Kaposi's sarcoma-associated herpesvirus G protein-coupled receptor. Cancer Cell 10 133 143

24. MoormanNJCristeaIMTerhuneSSRoutMPChaitBT 2008 Human cytomegalovirus protein UL38 inhibits host cell stress responses by antagonizing the tuberous sclerosis protein complex. Cell Host Microbe 3 253 262

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26. ClippingerAJMaguireTGAlwineJC 2011 The changing role of mTOR kinase in the maintenance of protein synthesis during human cytomegalovirus infection. J Virol 85 3930 3939

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PLOS Pathogens

2011 Číslo 9
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